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Forsíða

Forsíða

Óttar Rolfsson - Professor

Óttar Rolfsson

–

Heilbrigðisvísindasvið

Óttar Rolfsson

Professor

of Biochemistry and Systems Biology

Location
Sturlugata 8
Phone
525 - 5854
Email
ottarr [at] hi.is

Unit
Biochemistry
Website
https://systemsbiology.hi.is/the-rolfsson-laboratory/

ORCID site
https://orcid.org/0000-0003-4258-6057
Research portal – Óttar Rolfsson
Under construction throughout the autumn semester 2023

Published works

2023

Data from EMT-Derived Alterations in Glutamine Metabolism Sensitize Mesenchymal Breast Cells to mTOR Inhibition
Supplementary Figure 5 from EMT-Derived Alterations in Glutamine Metabolism Sensitize Mesenchymal Breast Cells to mTOR Inhibition
Endothelial Cell Phenotypes Demonstrate Different Metabolic Patterns and Predict Mortality in Trauma PatientsInternational Journal of Molecular Sciences
Supplementary Figure 1 from EMT-Derived Alterations in Glutamine Metabolism Sensitize Mesenchymal Breast Cells to mTOR Inhibition
Supplementary Figure 2 from EMT-Derived Alterations in Glutamine Metabolism Sensitize Mesenchymal Breast Cells to mTOR Inhibition
Supplementary Figure 3 from EMT-Derived Alterations in Glutamine Metabolism Sensitize Mesenchymal Breast Cells to mTOR Inhibition
Supplementary Figure 4 from EMT-Derived Alterations in Glutamine Metabolism Sensitize Mesenchymal Breast Cells to mTOR Inhibition
Supplementary File 1 from EMT-Derived Alterations in Glutamine Metabolism Sensitize Mesenchymal Breast Cells to mTOR Inhibition
Supplementary Figure 7 from EMT-Derived Alterations in Glutamine Metabolism Sensitize Mesenchymal Breast Cells to mTOR Inhibition
Supplementary Figure 4 from EMT-Derived Alterations in Glutamine Metabolism Sensitize Mesenchymal Breast Cells to mTOR Inhibition
Supplementary Figure 3 from EMT-Derived Alterations in Glutamine Metabolism Sensitize Mesenchymal Breast Cells to mTOR Inhibition
Supplementary Figure 6 from EMT-Derived Alterations in Glutamine Metabolism Sensitize Mesenchymal Breast Cells to mTOR Inhibition
Supplementary Figure 5 from EMT-Derived Alterations in Glutamine Metabolism Sensitize Mesenchymal Breast Cells to mTOR Inhibition
Supplementary Figure 2 from EMT-Derived Alterations in Glutamine Metabolism Sensitize Mesenchymal Breast Cells to mTOR Inhibition
Supplementary Figure 1 from EMT-Derived Alterations in Glutamine Metabolism Sensitize Mesenchymal Breast Cells to mTOR Inhibition
Supplementary File 1 from EMT-Derived Alterations in Glutamine Metabolism Sensitize Mesenchymal Breast Cells to mTOR Inhibition
Supplementary Figure 7 from EMT-Derived Alterations in Glutamine Metabolism Sensitize Mesenchymal Breast Cells to mTOR Inhibition
Supplementary Figure 6 from EMT-Derived Alterations in Glutamine Metabolism Sensitize Mesenchymal Breast Cells to mTOR Inhibition

2022

UDP‐glucose dehydrogenase expression is upregulated following EMT and differentially affects intracellular glycerophosphocholine and acetylaspartate levels in breast mesenchymal cell linesMolecular Oncology

2021

2020

2019

Pathogen inactivation with amotosalen plus UVA illumination minimally impacts microRNA expression in platelets during storage under standard blood banking conditionsTransfusion

2018

2017

2016

2015

2014

2013

2012

Inferring the metabolism of human orphan metabolites from their metabolic network context affirms human gluconokinase activityBiochem. J.

2011

The human metabolic reconstruction Recon 1 directs hypotheses of novel human metabolic functionsBMC Systems Biology

2010

2008

RNA Packing Specificity and Folding during Assembly of the Bacteriophage MS2Computational and Mathematical Methods in Medicine

2007

A Simple, RNA-Mediated Allosteric Switch Controls the Pathway to Formation of a T=3 Viral CapsidJournal of Molecular Biology

2003

Linkage of Osteoporosis to Chromosome 20p12 and Association to BMP2Plos Biol

Keywords for Specialisation:

Clinical Biochemistry

Cancer metabolism

Vascular endothelium